Vita C Infusion Mikrodermabrazja - Twoje Klucze do Promiennej Skóry

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4 / 5

25 marca 2023, o 19:48

Nie wie, czy kupi ponownie

Używa produktu od: miesiąc

Wykorzystała: jedno opakowanie

Kupiony w: Drogeria Rossmann

Pięknie pachnie.

Kosmetyk pachnie cytrusowo, dosyć intensywnie. Kosmetyk jest żółtawy. Na skórze czuć drobinki, które masują moją skórę. Zauważyłam lekkie rozjaśnienie mojej skóry. Peeling stosuję regularnie eaz q tygodniu. Nie czuję podrażnienia ani uczulenia. Nie należy mocno masować skóry twarzy ponieważ może zaistnieć zaczerwienienie.

Dodanych produktów: 25

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5 / 5

6 lutego 2021, o 11:11

Nie wie, czy kupi ponownie

Używa produktu od: kilka miesięcy

Wykorzystała: jedno opakowanie

Kupiony w: Nie podano

Bardzo fajny

Kupiłam ten peeling z przypadku i pozytywnie mnie zaskoczył.
Opakowanie proste, wygodne w użyciu.
Zapach delikatny, przyjemny.
Nie spowodował u mnie uczulenia ani wysuszenia.
Skóra po użyciu jest zdecydowanie wygładzona, czuć że została wypeelingowana. Nie jest pozdzierana. Konsystencja lekkiego kremu z malutkimi drobinkami ledwo wyczuwalnymi pod palcami, nie spodziewałam się że tak fajnie zadziała.

ZALETY: opakowanie
działanie
zapach

Dodanych produktów: 0

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5 / 5

14 listopada 2020, o 21:55

Używa produktu od: rok lub dłużej

Wykorzystała: jedno opakowanie

Rekindling vitamin C cancer therapy: oral vs intravenous administration

Based on studies pioneered by Mark Levine’s group at the NIH in the 2000s, the oral vitamin C doses used in the Mayo Clinic studies would have produced peak plasma concentration of less than 200 μM. In contrast, the same dose given intravenously, as used in the Pauling studies, would produce peak plasma concentrations of nearly 6 mM, more than 25 times higher. When given orally, vitamin C concentration in human plasma is tightly controlled by multiple mechanisms acting together: intestinal absorption, tissue accumulation, renal reabsorption and excretion, and potentially even the rate of utilization. However, when ascorbate is administered intravenously or intraperitoneally the tight controls are bypassed, and pharmacologic millimolar plasma concentrations of vitamin C can easily be achieved. For example, a phase I clinical study revealed that ascorbate concentrations could safely reach 25-30 mM with intravenous infusion of 100 g of vitamin C. In this study, plasma concentrations around 10 mM were sustained for at least 4 hours which, based on preclinical studies, is sufficient to kill cancer cells. Given the fact that cancer patients were only treated with vitamin C orally in the Mayo Clinic studies, the studies do not disprove high dose vitamin C’s efficacy as a cancer treatment.

More than half of colorectal cancers (CRCs) harbor activating mutations in KRAS or BRAF, yet those cancers are the most refractory to current targeted therapies. Our group and others showed that oncogenic mutations in KRAS or BRAF contribute to the Warburg effect and the addiction to glucose in part by upregulating a glucose transporter, GLUT1, that allows cancer cells to take up glucose efficiently. These data suggest a strategy for targeting KRAS or BRAF mutant cancer by exploiting the selective expression of GLUT1 and the metabolic liability that comes with increased reliance on glycolysis. Indeed, by targeting these unique features in these cancer cells, we recently showed that high dose vitamin C could selectively kill KRAS or BRAF mutant CRC cells.

How Does High-Dose Vitamin C Help Treat Cancer?

High-dose vitamin C has been studied as a treatment for patients with cancer since the 1970s. A Scottish surgeon named Ewan Cameron worked with Nobel Prize-winning chemist Linus Pauling to study the possible benefits of vitamin C therapy in clinical trials of cancer patients in the late 1970s and early 1980's.

Surveys of healthcare practitioners at United States CAM conferences in recent years have shown that high-dose IV vitamin C is frequently given to patients as a treatment for infections, fatigue , and cancers, including breast cancer.

More than fifty years ago, a study suggested that cancer was a disease of changes in connective tissue caused by a lack of vitamin C. In the 1970's, it was proposed that high-dose ascorbic acid could help build resistance to disease or infection and possibly treat cancer.

What Drugs Interact with High-Dose Vitamin C?

A drug interaction is a change in the way a drug acts in the body when taken with certain other drugs. High-dose vitamin C, when combined with some anticancer drugs, may cause them to be less effective. So far, these effects have been seen only in some laboratory and animal studies. No clinical trials have been done to further research these drug interactions in humans.

Laboratory studies and animal studies have been done to find out if high-dose vitamin C may be useful in preventing or treating cancer.

Laboratory studies

Many laboratory studies have been done to find out how high-dose vitamin C may cause the death of cancer cells. The anticancer effect of vitamin C in different types of cancer cells involves a chemical reaction that makes hydrogen peroxide, which may kill cancer cells.

Laboratory studies have shown the following:

• Treatment with high-dose vitamin C slowed the growth and spread of prostate, pancreatic, liver, colon, malignant mesothelioma, neuroblastoma, and other types of cancer cells.
• Combining high-dose vitamin C with certain types of chemotherapy may be more effective than chemotherapy alone:
  • Ascorbic acid with arsenic trioxide may be more effective in ovarian cancer cells.
  • Ascorbic acid with gemcitabine may be more effective in pancreatic cancer cells.
  • Ascorbic acid with gemcitabine and epigallocatechin-3-gallate (EGCG) may be more effective in malignant mesothelioma cells.

  However, not all laboratory studies combining vitamin C with anticancer therapies have shown benefit. Combining dehydroascorbic acid, a particular form of vitamin C, with chemotherapy made it less effective in killing some kinds of cancer cells.

  Animal studies

  Studies of high-dose vitamin C have been done in animal models (animals given a disease either the same as or like a disease in humans).

  

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