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Wyprysk z hiperkeratozą - Objawy, Diagnoza i Leczenie
A Word From Verywell
A skin condition can be challenging to deal with, especially if it causes painful symptoms. The good news is that dealing with most forms of hyperkeratosis is manageable with the proper treatment. In most cases, this condition is not severe or life-threatening.
The best thing you can do if you have hyperkeratosis is to speak to a dermatologist (a medical doctor specializing in conditions of the skin, hair, and nails) about your condition and any concerns you have. They will be able to determine the next steps to address your condition.
Frequently Asked Questions
The treatment for hyperkeratosis will depend entirely on its type and the underlying cause. Treatment isn't always necessary because some forms of hyperkeratosis are either asymptomatic or present with mild cosmetic symptoms. The most common forms of treatment include keratolytics, moisturizers, emollients, and retinoids.
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- Jakeman A. The effective management of hyperkeratosis.Wounds Int. 2012,1:65-73.
- National Human Genome Research Institute. Genetic disorders.
- Tian Y, Li XX, Zhang JJ, Yun Q, Zhang S, Yu JY, Feng XJ, Xia AT, Kang Y, Huang F, Wan F. Clinical outcomes and 5-year follow-up results of keratosis pilaris treated by a high concentration of glycolic acid.World J Clin Cases. 2021 Jun 26,9(18):4681-4689. doi:10.12998/wjcc.v9.i18.4681
- Harvard Health Publishing Harvard Medical School. Hyperkeratosis.
- National Organization for Rare Disorders. Lamellar Ichthyosis.
By Angelica Bottaro
Bottaro has a Bachelor of Science in Psychology and an Advanced Diploma in Journalism. She is based in Canada.
Hiperkeratoza to nie tylko nadmierne rogowacenie naskórka. Może objawiać się także poprzez powstawanie nagniotków, odcisków czy brodawek oraz łuszczenie się skóry. W hiperkeratozie na podeszwach stóp powstają również modzele. Są to płaskie obszary zrogowaciałej skóry, które mogą wywoływać dyskomfort podczas chodzenia, a nawet ból.
Hiperkeratoza pojawia się nie tylko na stopach. Niektóre osoby zmagają się z hiperkeratozą podpaznokciową. Jest ona objawem łuszczycy paznokci. Wywołuje ją rogowacenie naskórka pod paznokciem. Płytka paznokcia staje się krucha i podatna na złamania. Na jej powierzchni można dostrzec rowki i wgłębienia. Mówi się, że paznokieć zaczyna wyglądać jak naparstek. Z upływem czasu zmienia kolor i zaczyna odwarstwiać się od palca. Konieczne jest podjęcie leczenia, by móc cieszyć się zdrowymi paznokciami.
Również w innych rodzajach łuszczycy może pojawić się hiperkeratoza. Objawia się ona w formie grudek, które pokryte są łuską w kolorze srebrzystym. Występuje w różnych częściach ciała, np. na kolanach, łokciach, w okolicach pośladków czy na głowie.
Kiedy hiperkeratoza dotyka skóry głowy, staje się ona sucha i łuszczy się. Osoba chora może zauważyć łupież, a skóra staje się szorstka w dotyku. Kondycja włosów pogarsza się, mogą one zacząć wypadać. W takim przypadku skonsultuj z lekarzem, czy nie jest to czasem łuszczyca skóry głowy.
Histopathology
Psoriasis and psoriasiform dermatitis: It shows perivascular aggregates of lymphocytes in the dermal-epidermal junction with focal migration of leukocytes (neutrophils, lymphocytes) into the epidermis. There is increased epidermal proliferation and elongation of rete ridges giving an undulating appearance to the epidermis (papillomatosis) with or without spongiosis. The altered differentiation of keratinocytes results in hyperkeratosis with parakeratosis. Psoriasis also shows the formation of microabscesses by small aggregates of neutrophils in the upper epidermis (pustules) or in the stratum corneum (Munro microabscesses).
Interface and lichenoid dermatitis: Dense aggregates of lymphocytes along the dermal-epidermal junction associated with vacuolation of basal keratinocytes.[6] There is dyskeratosis, hyperkeratosis, and is sometimes associated with hypergranulosis.
Verrucae vulgaris and plana are characterized by marked hyperkeratosis, papillomatosis, and acanthosis. A typical feature is the presence of koilocytes, cells infected with papillomavirus which have structural changes like perinuclear halos and keratohyalin granules. Koilocytes can be absent in older lesions, but when present, are located in the upper stratum spinosum or granulosum. Parakeratosis may be present.
Seborrheic keratosis features marked hyperkeratosis, papillomatosis, and acanthosis. Pseudo-cysts and horn cysts are frequently present. There may be lymphocytic infiltrate and pigmentation as secondary features when irritated or inflamed.
The ichthyoses are a group of diseases caused by altered keratinization. The most common forms are ichthyosis vulgaris, X-linked, congenital, and epidermolytic hyperkeratosis.[7] They can be hereditary or acquired during life.[8][9] All of the forms show a defective epidermal barrier that induces hyperkeratosis, skin scaling, and inflammation.
Squamous cell carcinoma (SCC) is a neoplastic proliferation of atypical keratinocytes, restricted only to the epidermis (SCC in situ or Bowen's disease) or infiltrating the dermis (infiltrative SCC). Classic features are hyper-parakeratosis and loss of the granular layer.
StatPearls [Internet].
Fabiola Farci , Gauri D. Mahabal .
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Last Update: September 4, 2023 .
Hyperkeratosis refers to the increased thickness of the stratum corneum, the outer layer of the skin. It is most frequently due to chronic physical or chemical damage such as friction or the use of aggressive soaps but can also derive from chronic inflammation or a side-effect of different drugs, including chemotherapy. This activity reviews the evaluation and treatment of hyperkeratosis and highlights the role of the interprofessional team in evaluating and treating patients with this condition.
Summarize the major histologic forms of hyperkeratosis. Review the different pathologies that can manifest with hyperkeratosis.Explain the importance of collaboration and communication amongst the interprofessional team to ensure the appropriate diagnosis and treatment is selected for patients with hyperkeratosis.
